Advances in the mechanism of gallic acid regulating apoptosis in human cervical cancer HeLa cells

SONG Shaoting; LI Shuai; BAI Xue; FENG Caiyun; FAN Xiaoxia; LI Baoli

doi:10.16305/j.1007-1334.2020.1910049

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Advances in the mechanism of gallic acid regulating apoptosis in human cervical cancer HeLa cells

更新时间：2022-08-31

- Advances in the mechanism of gallic acid regulating apoptosis in human cervical cancer HeLa cells
- Shanghai Journal of Traditional Chinese Medicine Vol. 54, Issue 9, Pages: 99-101(2020)
- 作者机构：
  
  1. 延安大学医学院,陕西,延安,716000
  2. 陕西省延安市人民医院检验科,陕西,延安,716000
- 作者简介：
- 基金信息：
- DOI：10.16305/j.1007-1334.2020.1910049
  CLC：
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SONG Shaoting, LI Shuai, BAI Xue, et al. Advances in the mechanism of gallic acid regulating apoptosis in human cervical cancer HeLa cells. [J]. Shanghai Journal of Traditional Chinese Medicine 54(9):99-101(2020)
DOI：

SONG Shaoting, LI Shuai, BAI Xue, et al. Advances in the mechanism of gallic acid regulating apoptosis in human cervical cancer HeLa cells. [J]. Shanghai Journal of Traditional Chinese Medicine 54(9):99-101(2020) DOI： 10.16305/j.1007-1334.2020.1910049.

摘要

概述近年来没食子酸对人宫颈癌HeLa细胞凋亡机制的研究进展。没食子酸主要是通过调控P53信号通路、调节Caspase-3水平变化、促进ROS水平升高及调控ADAM17、EGFR、AKT/p-AKT和ERK/p-erk信号通路,实现对宫颈癌HeLa细胞凋亡的调控。

Abstract

To review the research progress of gallic acid on the mechanism of apoptosis of human cervical cancer HeLa cells in recent years. Gallic acid regulates the apoptosis of cervical cancer HeLa cells by regulating P53 signal pathway, regulating the change of Caspase-3 level, promoting the increase of ROS level and regulating ADAM17, EGFR, AKT/p-AKT and ERK/p-erk signal pathway.

关键词

没食子酸宫颈癌HeLa细胞凋亡综述

Keywords

gallic acidcervical cancerHeLa cellsapoptosisreview

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